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Periapical lesions decrease insulin signal and cause insulin resistance
(2013-05-01)
Introduction: Inflammatory cytokines are associated with decreased insulin signal transduction. Moreover, local oral inflammation, such as that accompanying periodontal disease, is associated with insulin resistance and ...
Insulin signaling proteins in pancreatic islets of insulin-resistant rats induced by glucocorticoid
(Soc Biolgia Chile, 2011-01-01)
Chronic administration of glucocorticoids induces insulin resistance that is compensated by an increase in beta-cell function and mass. Since insulin signaling is involved in the control of beta-cell function and mass, we ...
Insulin signaling proteins in pancreatic islets of insulin-resistant rats induced by glucocorticoid
(Soc Biolgia Chile, 2011-01-01)
Chronic administration of glucocorticoids induces insulin resistance that is compensated by an increase in beta-cell function and mass. Since insulin signaling is involved in the control of beta-cell function and mass, we ...
Insulin signaling proteins in pancreatic islets of insulin-resistant rats induced by glucocorticoid
(2011-11-15)
Chronic administration of glucocorticoids induces insulin resistance that is compensated by an increase in β-cell function and mass. Since insulin signaling is involved in the control of β-cell function and mass, we ...
Insulin signaling proteins in pancreatic islets of insulin-resistant rats induced by glucocorticoid
(Soc Biolgia Chile, 2014)
Challenges of the intensive insulin therapy in experimental models of extensive burn injury
(World Journal Series, 2020-08-30)
Insulin signal decrease in muscle but not in the liver of castrated male rats from chronic exposure to fluoride
(2010-01-01)
With half the animals as controls without fluoride (F) in their drinking water, 16 of 32 seven-week-old castrated male Wistar rats were administered NaF in their drinking water and F contained in food pellets (estimated ...
Lack of insulin inhibition on insulin secretion in non-diabetic morbidly obese patients
(Nature Publishing GroupBasingstokeInglaterra, 2001)