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Inflammation-induced modulation of cellular galectin-1 and-3 expression in a model of rat peritonitis
(Birkhauser Verlag Ag, 2014)
Inflammation-induced modulation of cellular galectin-1 and-3 expression in a model of rat peritonitis
(Birkhauser Verlag Ag, 2006-03-01)
Objective and design: To investigate the effect of galectin-1 (Gal-1) and -3 (Gal-3) on leukocyte migration and analyze the expression of both galectins in inflammatory cells using a model of rat peritonitis.Material or ...
Inflammation-induced modulation of cellular galectin-1 and-3 expression in a model of rat peritonitis
(Birkhauser Verlag Ag, 2006-03-01)
Objective and design: To investigate the effect of galectin-1 (Gal-1) and -3 (Gal-3) on leukocyte migration and analyze the expression of both galectins in inflammatory cells using a model of rat peritonitis.Material or ...
Quantum Dots Modulate Leukocyte Adhesion and Transmigration Depending on Their Surface Modification
(Amer Chemical SocWashingtonEUA, 2010)
SOCS3 expression correlates with severity of inflammation, expression of proinflammatory cytokines, and activation of STAT3 and p38 MAPK in LPS-induced inflammation in vivo
(2013-10-07)
SOCS3 is an inducible endogenous negative regulator of JAK/STAT pathway, which is relevant in inflammatory conditions. We used a model of LPS-induced periodontal disease in rats to correlate SOCS3 expression with the ...
SOCS3 expression correlates with severity of inflammation, expression of proinflammatory cytokines, and activation of STAT3 and p38 MAPK in LPS-induced inflammation in vivo
(2013-10-07)
SOCS3 is an inducible endogenous negative regulator of JAK/STAT pathway, which is relevant in inflammatory conditions. We used a model of LPS-induced periodontal disease in rats to correlate SOCS3 expression with the ...