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Alteration of sFAS and sFAS ligand expression during canine visceral leishmaniosis
(Elsevier B.V., 2014-10-15)
Visceral leishmaniosis (VL) is caused by intracellular parasites of the genus Leishmania that affect humans and several animal species. Dogs are one of the main urban reservoirs of Leishmania infantum and play a central ...
Alteration of sFAS and sFAS ligand expression during canine visceral leishmaniosis
(Elsevier B.V., 2015)
CD95 (FAS) and CD178 (FASL) induce the apoptosis of CD4+ and CD8+ cells isolated from the peripheral blood and spleen of dogs naturally infected with Leishmania spp
(2013-10-08)
Infected dogs are urban reservoirs of Leishmania chagasi, which is a causative agent of visceral leishmaniasis (VL). Dogs exhibit immune suppression during the course of this disease, and lymphocyte apoptosis is involved ...
CD95 (FAS) and CD178 (FASL) induce the apoptosis of CD4+ and CD8+ cells isolated from the peripheral blood and spleen of dogs naturally infected with Leishmania spp
(2013-10-08)
Infected dogs are urban reservoirs of Leishmania chagasi, which is a causative agent of visceral leishmaniasis (VL). Dogs exhibit immune suppression during the course of this disease, and lymphocyte apoptosis is involved ...
The role of Fas ligand protein in the oxidative stress induced by azoxymethane on crypt colon of rats
(Acta Cirurgica Brasileira, 2010-11-01)
Purpose: To study the protein Fas ligand (FasL) on the expression of apoptosis, using a model of oxidative stress induced by azoxymethane (AOM), in the crypt of colon in rats. Methods: Wistar rats (n=14) were assigned into ...
Quantification of Fas protein in CSF of patients with neurocysticercosis
(Academia Brasileira de Neurologia - ABNEURO, 2012-04-01)
Neurocysticercosis is a parasitic disease that affects the central nervous system. The objective of this study was to investigate the correlation between neuronal death evaluated by the quantification of Fas apoptotic ...
Alteration of sFAS and sFAS ligand expression during canine visceral leishmaniosis
(Elsevier B.V., 2015)