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Cardiac Thyrotropin-releasing Hormone Inhibition Improves Ventricular Function and Reduces Hypertrophy and Fibrosis After Myocardial Infarction in Rats
(Churchill Livingstone Inc Medical Publishers, 2021-07)
Background: Cardiac thyrotropin-releasing hormone (TRH) is a tripeptide with still unknown functions. We demonstrated that the left ventricle (LV) TRH system is hyperactivated in spontaneously hypertensive rats and its ...
Transient Receptor Potential Canonical 3 and Nuclear Factor of Activated T Cells C3 Signaling Pathway Critically Regulates Myocardial Fibrosis
(Mary Ann Liebert, 2019-06)
Aims: Cardiac fibroblasts (CFs) are emerging as major contributors to myocardial fibrosis (MF), a final common pathway of many etiologies of heart disease. Here, we studied the functional relevance of transient receptor ...
Low-intensity aerobic exercise improves cardiac remodelling of adult spontaneously hypertensive rats
(2019-01-01)
We evaluated the influence of aerobic training on cardiac remodeling in untreated spontaneously hypertensive rats (SHR). Four experimental groups were used: sedentary (W-SED, n=27) and trained (WEX, n=31) normotensive ...
Long-term low intensity physical exercise attenuates heart failure development in aging spontaneously hypertensive rats
(Karger, 2015-01-01)
Background: Physical exercise is a strategy to control hypertension and attenuate pressure overload-induced cardiac remodeling. The influence of exercise on cardiac remodeling during uncontrolled hypertension is not ...
Myocardial edema without fibrosis by magnetic resonance T2 mapping in acute Chagas' myocarditis
(Sociedade Brasileira de Cardiologia, 2017)
Myocardial edema without fibrosis by magnetic resonance T2 mapping in acute Chagas' myocarditis
(Sociedade Brasileira de Cardiologia, 2019)
Isoproterenol and angiotensin I-converting enzyme in lung, left ventricle, and plasma during myocardial hypertrophy and fibrosis
(LIPPINCOTT WILLIAMS & WILKINS, 2002-08)
This study investigated whether long-term administration of isoproterenol (ISO) induces differential expression of angiotensin-converting enzyme (ACE) in lung, plasma, and left ventricle (LV) during development of left ...
Early rather than delayed administration of lisinopril protects the heart after myocardial infarction in rats
(Dr Dietrich Steinkopff Verlag, 2000-06-01)
Background: ACE inhibitors have shown beneficial results in several studies after myocardial infarction (MI). However, these studies have shown conflicting results about the ideal starting time of the ACE inhibitors ...
Early rather than delayed administration of lisinopril protects the heart after myocardial infarction in rats
(Dr Dietrich Steinkopff Verlag, 2000-06-01)
Background: ACE inhibitors have shown beneficial results in several studies after myocardial infarction (MI). However, these studies have shown conflicting results about the ideal starting time of the ACE inhibitors ...