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Identification of galectins as novel regulators of platelet signaling and function
(John Wiley & Sons Inc, 2011-07)
Platelet activation at sites of vascular injury leads to the formation of a hemostatic plug. Activation of platelets is therefore crucial for normal hemostasis. However, uncontrolled platelet activation may also lead to ...
Glycobiology of platelet–endothelial cell interactions
(Oxford University Press, 2014-06)
Under normal conditions, platelets do not interact with blood vessel walls; however, upon activation, platelets firmly attach to endothelial cells. Communication between platelets and endothelial cells during the normal ...
Current viewpoints on platelet contribution to inflammation
(Baishideng Publishing Group Inc, 2012-12)
Inflammation is an underlying feature of a variety of human diseases. Because inflammatory diseases are a major cause of morbidity and mortality in developed countries, understanding the interaction of the most important ...
The role of sex steroids on cellular events involved in vascular disease
(Pergamon-Elsevier Science Ltd, 2012-11)
In this work we checked the hypothesis whether estrone, progesterone, and testosterone are able to modulate the interactions between platelets, monocytes, and endothelial cells either under basal or inflammatory conditions. ...
Functional responses and molecular mechanisms involved in histone-mediated platelet activation
(Schattauer Gmbh-Verlag Medizin Naturwissenschaften, 2013-11)
Histones are highly alkaline proteins found in cell nuclei and they can be released by either dying or inflammatory cells. The recent observations that histones are major components of neutrophil extracellular traps and ...
The role of superoxide anion in the inhibitory effect of SIN-1 in thrombin-activated human platelet adhesion
(Elsevier Science BvAmsterdamHolanda, 2010)
Interaction of Human Platelets with Enterohemorrhagic E.coli O111, Induces an Increase in the Procoagulant Activity,Thrombin Generation and Adhesion to Endothelial Cells
(2014)
Acute inflammation in response to severe bacterial infection, results in
hemostatic abnormalities ranging from subclinical to sustained systemic
clotting activation leading to massive thrombin and fibrin formation ...