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Ex Vivo Treatment with a Polyphenol-Enriched Cocoa Extract Ameliorates Myocardial Infarct and Postischemic Mitochondrial Injury in Normotensive and Hypertensive Rats
(American Chemical Society, 2016-06)
Our objective was to determine the effects of a polyphenol-enriched cocoa extract (PCE) on myocardial postischemic alterations in normotensive (Wistar rats, W) and spontaneously hypertensive rats (SHR). Isolated hearts ...
Long-term effects of growth hormone on infarct size and left ventricular function in sheep with coronary artery occlusion
(Lippincott Williams, 2010-03)
The effects of growth hormone (GH) on infarct size and left ventricular (LV) function in experimental acute myocardial infarction (AMI) have been controversial. Moreover, little, if any, information exists regarding long-term ...
Targeting the Endothelium to Achieve Cardioprotection
(Frontiers Media, 2021)
Despite considerable improvements in the treatment of myocardial infarction, it is still a highly prevalent disease worldwide. Novel therapeutic strategies to limit infarct size are required to protect myocardial function ...
The effectiveness of antioxidant vitamins C and E in reducing myocardial infarct size in patients subjected to percutaneous coronary angioplasty (PREVEC Trial): study protocol for a pilot randomized double-blind controlled trial
(BioMed Central, 2014)
Background: Acute myocardial infarction (AMI) is the leading cause of mortality worldwide. Oxidative stress has
been involved in the ischemia-reperfusion injury in AMI. It has been suggested that reperfusion accounts for ...
Mesenchymal stromal cells overexpressing vascular endothelial growth factor in ovine myocardial infarction
(Nature Publishing Group, 2015-06)
Mesenchymal stromal cells (MSCs) are cardioprotective in acute myocardial infarction (AMI). Besides, we have shown that intramyocardial injection of plasmid-VEGF165 (pVEGF) in ovine AMI reduces infarct size and improves ...
Changes in the loading conditions induced by vagal stimulation modify the myocardial infarct size through sympathetic-parasympathetic interactions
(Springer, 2015-07)
In a previous research, we described that vagal stimulation increases the infarct size by sympathetic co-activation. The aim of this study was to determine if hemodynamic changes secondary to the vagal stimulation are able ...
Cardioprotective efficacy against reperfusion injury of EMD-87580: Comparison to ischemic postconditioning
(Elsevier Science, 2014-05-22)
Previous results show that prolonged treatment with EMD-87580 (EMD) NHE-1 blocker attenuates and reverses postinfarction remodelling. Our aim was to evaluate the effects of the treatment of EMD compared to ischemic ...