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Infarto del miocardio y tendinitis aquiliana como manifestaciones de hipercolesterolemia familiar
(Universidad de Antioquia, Facultad de MedicinaMedellín, Colombia, 2021)
Caracterización de pacientes con infarto agudo del miocardio sin enfermedad coronaria obstructiva
(Sociedad de Cirugía de Bogotá, Hospital de San José y Fundación Universitaria de Ciencias de la Salud, 2017-03-01)
Estudio descriptivo sobre bloqueos atrio-ventriculares en infartos posteroinferiores en el Hospital de Caldas ESE.
(2013-10-29)
Introducción: El infarto agudo de miocardio (IAM) es un problema de salud pública a escala mundial y nacional, que merece
una prevención, diagnóstico y tratamiento oportunos. El IAM de cara inferior presenta entre sus ...
The action of ovarian hormones in cardiovascular disease
(Sociedad de Biología de Chile, 2003)
The incidence of cardiovascular disease (CAD) differs between men and women, in part
because of differences in risk factors and hormones. This sexual dimorphism means a lower incidence in atherosclerotic diseases in ...
Infarto agudo al miocardio en pacientes de 80 y más años. Evolución hospitalaria y seguimiento
(Sociedad Médica de Santiago, 2008-03)
Subjects older than 80 years have more complicated
acute myocardial infarctions and are subjected to less reperfusion procedures. Aim: To describe
the hospital evolution and follow up of patients older than 80 years, ...
Dexmedetomidine preconditioning activates pro-survival kinases and attenuates regional ischemia/reperfusion injury in rat heart
(ELSEVIER, 2012-04)
Pharmacological preconditioning limits myocardial infarct size after ischemia/reperfusion. Dexmedetomidine is an alpha(2)-adrenergic receptor agonist used in anesthesia that may have cardioprotective properties against ...
Mitochondrial dynamics, mitophagy and cardiovascular disease
(WILEY-BLACKWELL, 2016)
Cardiac hypertrophy is often initiated as an adaptive response to haemodynamic stress or myocardial injury, and allows the heart to meet an increased demand for oxygen. Although initially beneficial, hypertrophy can ...
Angiotensin-(1-9) regulates cardiac hypertrophy in vivo and in vitro
(Lippincott Williams and Wilkins, 2010)
Background: Angiotensin-(1-9) is present in human and rat plasma and its circulating levels increased early after myocardial infarction or in animals treated with angiotensin-converting enzyme inhibitor. However, the ...
Studies on the role of apoptosis after transient myocardial ischemia: genetic deletion of the executioner caspases-3 and -7 does not limit infarct size and ventricular remodeling
(Springer, 2016)
Although it is widely accepted that apoptosis
may contribute to cell death in myocardial infarction,
experimental evidence suggests that adult cardiomyocytes
repress the expression of the caspase-dependent apoptotic
pathway. ...