The twisted survivin connection to angiogenesis

Molecular Cancer

dc.creatorSanhueza, C
dc.creatorWehinger, Sergio
dc.creatorCastillo-Bennett, Jimena
dc.creatorValenzuela, Manuel alejandro
dc.creatorOwen-Jardine, Gareth Ivor
dc.creatorQuest, A F G
dc.date2018-11-29T15:37:00Z
dc.date2022-07-07T15:58:37Z
dc.date2014
dc.date2018-11-29T15:37:00Z
dc.date2022-07-07T15:58:37Z
dc.date2015
dc.date.accessioned2023-08-22T22:38:30Z
dc.date.available2023-08-22T22:38:30Z
dc.identifier1140970
dc.identifier1140970
dc.identifierhttps://hdl.handle.net/10533/228609
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/8347796
dc.descriptionSurvivin, a member of the inhibitor of apoptosis family of proteins (IAPs) that controls cell division, apoptosis, metastasis and angiogenesis, is overexpressed in essentially all human cancers. As a consequence, the gene/protein is considered an attract
dc.descriptionRegular
dc.descriptionFONDECYT
dc.descriptionFONDECYT
dc.languageeng
dc.relationhandle/10533/111556
dc.relationhandle/10533/111541
dc.relationhandle/10533/108045
dc.relationhttps://www.researchgate.net/publication/284180117_The_twisted_survivin_connection_to_angiogenesis
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titleThe twisted survivin connection to angiogenesis
dc.titleMolecular Cancer
dc.typeArticulo
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion


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