Plos One

dc.creatorBustos, F.
dc.creatorVarela-Nallar, L.
dc.creatorCampos, M.
dc.creatorHenriquez, B.
dc.creatorPhilips, M.
dc.creatorOpazo, C.
dc.creatorAguayo, L. G.
dc.creatorMontecino, M.
dc.creatorConstantine-Paton, M.
dc.creatorInestrosa, N. C.
dc.creatorvan Zundert, B.
dc.date2020-03-11T20:25:39Z
dc.date2022-07-07T23:42:25Z
dc.date2020-03-11T20:25:39Z
dc.date2022-07-07T23:42:25Z
dc.date2014
dc.date.accessioned2023-08-22T01:47:12Z
dc.date.available2023-08-22T01:47:12Z
dc.identifier15090007
dc.identifier15090007
dc.identifierhttps://hdl.handle.net/10533/237850
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/8307614
dc.descriptionConsiderable evidence indicates that the NMDA receptor (NMDAR) subunits NR2A and NR2B are critical mediators of synaptic plasticity and dendritogenesis; however, how they differentially regulate these processes is unclear. Here we investigate the roles of
dc.descriptionFONDAP
dc.descriptionFONDAP
dc.languageeng
dc.relationhttps://doi.org/10.1371/journal.pone.0094037
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.rightsinfo:eu-repo/semantics/openAccess
dc.titlePSD-95 suppresses dendritic arbor development in mature hippocampal neurons by occluding the clustering of NR2B-NMDA receptors
dc.titlePlos One
dc.typeArticulo
dc.typeinfo:eu-repo/semantics/article
dc.typeinfo:eu-repo/semantics/publishedVersion


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