BCL-2 family: integrating stress responses at the ER to control cell demise

dc.date.accessioned2020-03-11T20:34:17Z
dc.date.accessioned2022-10-18T22:57:42Z
dc.date.available2020-03-11T20:34:17Z
dc.date.available2022-10-18T22:57:42Z
dc.date.created2020-03-11T20:34:17Z
dc.date.issued2017
dc.identifierhttp://hdl.handle.net/10533/240216
dc.identifier15150012
dc.identifierWOS:000407506500004
dc.identifierno scielo
dc.identifiereid=2-s2.0-85031314980
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4471555
dc.description.abstractIn the last decade, the endoplasmic reticulum (ER) has emerged as a central organelle regulating the core mitochondrial apoptosis pathway. At the ER membrane, a variety of stress signals are integrated toward determining cell fate, involving a complex cro
dc.languageeng
dc.relationhttps://doi.org/10.1038/cdd.2017.82
dc.relation10.1038/cdd.2017.82
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleBCL-2 family: integrating stress responses at the ER to control cell demise
dc.typeArticulo


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