dc.date.accessioned2018-11-29T15:35:52Z
dc.date.accessioned2022-10-18T21:23:19Z
dc.date.available2018-11-29T15:35:52Z
dc.date.available2022-10-18T21:23:19Z
dc.date.created2018-11-29T15:35:52Z
dc.date.issued2016
dc.identifierhttp://hdl.handle.net/10533/227986
dc.identifier1140325
dc.identifierWOS:000383314500016
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4459342
dc.description.abstractAxonal growth cone collapse following spinal cord injury (SCI) is promoted by semaphorin3A (Sema3A) signaling via PlexinA4 surface receptor. This interaction triggers intracellular signaling events leading to increased hydrogen peroxide levels which in t
dc.languageeng
dc.relationhttps://www.sciencedirect.com/science/article/pii/S001448861630173X
dc.relationhandle/10533/111556
dc.relation10.1016/j.expneurol.2016.06.009
dc.relationhandle/10533/111541
dc.relationhandle/10533/108045
dc.rightsinfo:eu-repo/semantics/openAccess
dc.rightsAtribución-NoComercial-SinDerivadas 3.0 Chile
dc.rightshttp://creativecommons.org/licenses/by-nc-nd/3.0/cl/
dc.titleLigand-mediated galectin-1 endocytosis prevents intraneural h2o2 production promoting f-actin dynamics reactivation and axonal re-growth
dc.typeArticulo


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