dc.creatorOrtiz Ramírez, Mario Isidoro
dc.date.accessioned2013-11-05T22:00:09Z
dc.date.accessioned2022-10-14T15:34:04Z
dc.date.available2013-11-05T22:00:09Z
dc.date.available2022-10-14T15:34:04Z
dc.date.created2013-11-05T22:00:09Z
dc.date.issued2008
dc.identifierChávez-Piña AE, Vong L, McKnight W, Dicay M, Zanardo RC, Ortiz MI, Castañeda-Hernández G, Wallace JL. Lack of effects of acemetacin on signalling pathways for leukocyte adherence may explain its gastrointestinal safety. Br J Pharmacol. 2008;155(6):857-64. ISSN:1476-5381.
dc.identifierhttp://repository.uaeh.edu.mx/bitstream/handle/123456789/11893
dc.identifier.urihttps://repositorioslatinoamericanos.uchile.cl/handle/2250/4254051
dc.description.abstractAcemetacin is a non-steroidal anti-inflammatory drug which is rapidly bioconverted to indomethacin, but produces significantly less gastric damage than indomethacin. This study was performed to investigate several possible mechanisms that could account for the gastrointestinal tolerability of acemetacin.
dc.subjectFarmacología de la Reproducción y Analgesia inducida por neuroesteroides y AINES
dc.titleLack of effects of acemetacin on signalling pathways for leukocyte adherence may explain its gastrointestinal safety
dc.typeArtículos de revistas


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