dc.creatorDe Almeida O.P.
dc.creatorBohm G.M.
dc.date1979
dc.date2015-06-30T12:53:50Z
dc.date2015-11-26T14:35:13Z
dc.date2015-06-30T12:53:50Z
dc.date2015-11-26T14:35:13Z
dc.date.accessioned2018-03-28T21:38:37Z
dc.date.available2018-03-28T21:38:37Z
dc.identifier
dc.identifierJournal Of Pathology. , v. 127, n. 1, p. 27 - 34, 1979.
dc.identifier223417
dc.identifier
dc.identifierhttp://www.scopus.com/inward/record.url?eid=2-s2.0-0018408354&partnerID=40&md5=57720196b86697b3adb2af5efc1db785
dc.identifierhttp://www.repositorio.unicamp.br/handle/REPOSIP/97761
dc.identifierhttp://repositorio.unicamp.br/jspui/handle/REPOSIP/97761
dc.identifier2-s2.0-0018408354
dc.identifier.urihttp://repositorioslatinoamericanos.uchile.cl/handle/2250/1248310
dc.descriptionThe vascular permeability of the vessels of clinically normal gingiva of rats was studied using the colloidal carbon technique. The connective tissue situated underneath the keratinized epithelium was normal, but that subjacent to the non-keratinized epithelium showed some degree of chronic inflammation and as a rule the vessels of the area exhibited increased vascular permeability. In the buccal gingiva, the vessels labeled with carbon form loops are situated 200 μm below the marginal gingiva, while in the interdental gingiva the altered vessels are just below the superficial epithelium. On electron microscopy, the vessels in the inflamed areas showed many open endothelial junctions, and also pseudo-fenestration, and endoplasmic vesicles full of carbon. Little is known about vascular changes in chronic inflammation, and the rat gingiva seems to be a suitable model for their study.
dc.description127
dc.description1
dc.description27
dc.description34
dc.languageen
dc.publisher
dc.relationJournal of Pathology
dc.rightsfechado
dc.sourceScopus
dc.titleVascular Permeability In The Rat Gingiva. A Model Of Vessel Response In Chronic Inflammation
dc.typeArtículos de revistas


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