Now showing items 11-20 of 503
Hyperkalemic periodic paralysis M1592V mutation modifies activation in human skeletal muscle Na+ channel
Mutations in the human skeletal muscle Na+ channel underlie the autosomal dominant disease hyperkalemic periodic paralysis (HPP). Muscle fibers from affected individuals exhibit sustained Na+ currents thought to depolarize ...
Effects of beta-adrenoceptor antagonists in the neural nitric oxide release induced by electrical field stimulation and sodium channel activators in the rabbit corpus cavemosum
(Elsevier Science BvAmsterdamHolanda, 2005)
Molecular determinants of binding of a wasp toxin (PMTXs) and its analogs in the Na+ channels proteins
(Elsevier B.V., 2000-05-05)
The structural specificity of alpha-PMTX, a novel peptide toxin derived from wasp venom has been studied on the neuromuscular synapse in the walking leg of the lobster. alpha-PMTX is known to induce repetitive action ...
ABNORMAL EXPRESSION OF VOLTAGE-GATED SODIUM CHANNELS Nav1.7, Nav1.3 AND Nav1.8 IN TRIGEMINAL NEURALGIA
(PERGAMON-ELSEVIER SCIENCE LTD, 2009)
Voltage-gated sodium channels have been implicated in acute and chronic neuropathic pain. Among subtypes, Nav1.7 single mutations can cause congenital indifference to pain or chronic neuropathic pain syndromes, including ...
Altered iPSC-derived neurons' sodium channel properties in subjects with Monge's disease
Monge's disease, also known as chronic mountain sickness (CMS), is a disease that potentially threatens more than 140 million highlanders during extended time living at high altitudes (over 2500m). The prevalence of CMS ...
Histochemical differences in the responses of predominantly fast-twitch glycolytic muscle and slow-twitch oxidative muscle to veratrine
(Pergamon-elsevier Science LtdOxfordInglaterra, 2002)
Crystal structure of neurotoxin Ts1 from Tityus serrulatus provides insights into the specificity and toxicity of scorpion toxins
(Academic Press LtdLondonInglaterra, 1999)