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Aldose reductase induced by hyperosmotic stress mediates cardiomyocyte apoptosis - Differential effects of sorbitol and mannitol
(AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC, 2003-10-03)
Cells adapt to hyperosmotic conditions by several mechanisms, including accumulation of sorbitol via induction of the polyol pathway. Failure to adapt to osmotic stress can result in apoptotic cell death. In the present ...
Regulation of intracellular pH is altered in cardiac myocytes of ovariectomized rats
(American Heart Association, Inc, 2019-04-02)
Background-—It is well known that after menopause women are exposed to a greater cardiovascular risk, but the intracellular modifications are not properly described. The sodium/proton exchanger (NHE) and the sodium/bicarbonate ...
Trypanosoma cruzi induces changes in cardiac connexin43 expression
(Elsevier, 2018)
Ca2+/ Calmodulin dependent protein kinase II and androgen signaling pathways modulate MEF2 activity in testosterone induced cardiac myocyte hypertrophy
(Frontiers media SA, 2017)
Testosterone is known to induce cardiac hypertrophy through androgen receptor (AR)-dependent and -independent pathways, but the molecular underpinnings of the androgen action remain poorly understood. Previous work has ...
Measuring [Ca2+] with fluorescent indicators: theoretical approach to the ratio method
(Churchill LivingstoneEdinburghEscócia, 1998)
Targeting focal adhesion kinase with small interfering RNA prevents and reverses load-induced cardiac hypertrophy in mice
(Lippincott Williams & WilkinsPhiladelphiaEUA, 2007)
Action potential duration determines sarcoplasmic reticulum Ca2+ reloading in mammalian ventricular myocytes
(Blackwell Publishing LtdOxfordInglaterra, 2004)
Insulin-like growth factor-I rapidly activates multiple signal transduction pathways in cultured rat cardiac myocytes
(1997)
In response to insulin-like growth factor-I (IGF-I), neonatal rat cardiac myocytes exhibit a hypertrophic response. The elucidation of the IGF- I signal transduction system in these cells remains unknown. We show here that ...