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Novel and optimized strategies for inducing fibrosis in vivo: focus on duchenne muscular dystrophy

Unknown author (BIOMED CENTRAL, 2014)

Novel and optimized strategies for inducing fibrosis in vivo: focus on duchenne muscular dystrophy

Unknown author (BIOMED CENTRAL, 2014)

Wnt signaling pathway improves central inhibitory synaptic transmission in a mouse model of Duchenne muscular dystrophy

Unknown author (2016)

Transforming growth factor type-beta inhibits Mas receptor expression in fibroblasts but not in myoblasts or differentiated myotubes; Relevance to fibrosis associated to muscular dystrophies

Unknown author (2015)

Novel and optimized strategies for inducing fibrosis in vivo: focus on Duchenne Muscular Dystrophy

Unknown author (2014)

Novel regulatory mechanisms for the proteoglycans decorin and biglycan during muscle formation and muscular dystrophy

Unknown author (2008)

Novel regulatory mechanisms for the proteoglycans decorin and biglycan during muscle formation and muscular dystrophy

Unknown author (2008)

s/i

Increase in decorin and biglycan in duchenne muscular dystrophy: role of fibroblasts as cell source of these proteoglycans in the disease

Unknown author (FOUNDATION FOR CELLULAR AND MOLECULAR MEDICINE, 2006)

Augmented synthesis and differential localization of heparan sulfate proteoglycans in duchenne muscular dystrophy

Unknown author (WILEY-LISS, 2002)

Muscle MRI in a large cohort of patients with oculopharyngeal muscular dystrophy

Unknown author (2019)

Background and objective Oculopharyngeal muscular dystrophy (OPMD) is a genetic disorder caused by an abnormal expansion of GCN triplets within the PABPN1 gene. Previous descriptions have focused on lower limb muscles in ...

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