Now showing items 1-10 of 50
Differences in both inositol 1,4,5-triphosphate mass and inositol 1,4,5- triphosphate receptors between normal and dystrophic between normal and dystrophic skeletal muscle cell lines
Human normal (RCMH) and Duchenne muscular dystrophy (RCD) cell lines, as well as newly developed normal and dystrophic murine cell lines, were used for the study of both changes in inositol 1,4,5-trisphosphate (IP3) mass ...
INOSITOL TRIPHOSPHATE PARTICIPATES IN AN OESTRADIOL NONGENOMIC SIGNALLING PATHWAY INVOLVED IN ACCELERATED OVIDUCTAL TRANSPORT IN CYCLING RATS
(ASSOCIATION FOR THE STUDY OF INTERNAL SECRETIONS, 2006)
Mensageiros secundários relacionados à ação dos hormônios vegetais
The current knowledge about the mechanisms of action of plant hormones is still fragmented and incomplete. However, it’s known that the regulatory action and that the potential of signal amplification of these molecules ...
MTOR-independent autophagy induced by interrupted endoplasmic reticulum-mitochondrial Ca2+ communication: a dead end in cancer cells
(Taylor and Francis Inc., 2019)
© 2018, © 2018 Informa UK Limited, trading as Taylor & Francis Group. The interruption of endoplasmic reticulum (ER)-mitochondrial Ca2+ communication induces a bioenergetic crisis characterized by an increase of MTOR-independent ...
Desensitization of Angiotensin II effect on [Ca2+]i, inositol triphosphate and prolactin in pituitary cells
(American Physiological Society, 2001-12)
Activation of pituitary angiotensin (ANG II) type 1 receptors (AT1) mobilizes intracellular Ca2+, resulting in increased prolactin secretion. We first assessed desensitization of AT1 receptors by testing ANG II-induced ...
Inositol trisphosphate and excitation-contraction coupling in skeletal muscle
(Kluwer Academic Publishers-Plenum Publishers, 1989)
The role of inositol trisphosphate as a chemical messenger in excitation-contraction coupling is discussed, both in terms of positive and negative results. The evidence presented includes experiments on the effect of ...
Alteration in mitochondrial Ca2+ uptake disrupts insulin signaling in hypertrophic cardiomyocytes
(BioMed Central Ltd., 2014)
© 2014 Gutierrez et al.; licensee BioMed Central Ltd. Background: Cardiac hypertrophy is characterized by alterations in both cardiac bioenergetics and insulin sensitivity. Insulin promotes glucose uptake by cardiomyocytes ...