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Diverse effects of renal denervation on ventricular hypertrophy and blood pressure in DOCA-salt hypertensive rats
(1998-04)
Cardiac hypertrophy that accompanies hypertension seems to be a phenomenon of multifactorial origin whose development does not seem to depend on an increased pressure load alone, but also on local growth factors and ...
Coronary vascular and myocardial lesions due to experimental constriction of the abdominal aorta
(1992-01-01)
The development and evolution of arterial and myocardial lesions were morphologically evaluated in Wistar rats submitted to constriction of the abdominal aorta. The control (sham-operated) and operated groups were evaluated ...
Macrophage migration inhibitory factor in the nucleus of solitary tract decreases blood pressure in SHRs
(2013-01-01)
Aims The macrophage migration inhibitory factor (MIF) is an intracellular inhibitor of the central nervous system actions of angiotensin II on blood pressure. Considering that angiotensin II actions at the nucleus of the ...
Macrophage migration inhibitory factor in the nucleus of solitary tract decreases blood pressure in SHRs
(2013-01-01)
Aims The macrophage migration inhibitory factor (MIF) is an intracellular inhibitor of the central nervous system actions of angiotensin II on blood pressure. Considering that angiotensin II actions at the nucleus of the ...
A role for focal adhesion kinase in cardiac mitochondrial biogenesis induced by mechanical stress
(Amer Physiological SocBethesdaEUA, 2011)
Cardioprotective mechanism of S-nitroso-N-acetylcysteine via S-nitrosated betadrenoceptor-2 in the LDLr-/- mice
(Academic Press Inc Elsevier ScienceSan DiegoEUA, 2014)
The effect of non-antihypertensive doses of angiotensin converting enzyme inhibitor on myocardial necrosis and hypertrophy in young rats with renovascular hypertension
(1999-05-06)
In renovascular hypertensive rats, low doses of angiotensin converting enzyme (ACE) inhibitors have been found to prevent myocardial hypertrophy independent of blood pressure level. This finding would suggest humoral rather ...
MEF2C Silencing Attenuates Load-Induced Left Ventricular Hypertrophy by Modulating mTOR/S6K Pathway in Mice
(Public Library ScienceSan FranciscoEUA, 2009)