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Inactivación del gen CDKN2A (p16) en cáncer de la vesícula biliar
(Sociedad Médica de Santiago, 2004)
DNA methylation analysis of the tumor suppressor gene CDKN2B in Brazilian leukemia patients
(Sociedade Brasileira de Genética, 2008)
The aim of this work was to evaluate the methylation profile of the p15 (CDKN2B) gene in Brazilian patients with leukemia and to correlate the CDKN2B gene expression with the percentage of methylated CpG dinucleotides in ...
Gene Mutations in Esophageal Mucosa of Chagas Disease Patients
(Int Inst Anticancer Research, 2009-04-01)
Background: Chagas disease patients with longstanding megaesophagus have a significantly increased risk for esophageal carcinoma. Materials and Methods: PCR-SSCP analysis and DNA sequencing of esophageal mucosa from Chagas ...
Gene Mutations in Esophageal Mucosa of Chagas Disease Patients
(Int Inst Anticancer Research, 2014)
Methylation status of ANAPC1, CDKN2A and TP53 promoter genes in individuals with gastric cancer
(Associação Brasileira de Divulgação Científica, 2008)
Gastric cancer is the forth most frequent malignancy and the second most common cause of cancer death worldwide. DNA methylation is the most studied epigenetic alteration, occurring through a methyl radical addition to the ...
Estudio molecular de los genes CDKN1B y CITED2 en mujeres con falla ovárica prematura idiopática
(Universidad del RosarioMaestría en Ciencias con Énfasis en Genética HumanaFacultad de medicina, 2011)
INACTIVATION OF CDKN2A GENE (P16) IN GALLBLADDER CARCINOMA
(SOCIEDAD MEDICA DE SANTIAGO, 2004)
CDKN2A promoter hypermethylation in astrocytomas is associated with age and sex
(2013-06-12)
CDKN2A promoter hypermethylation has been widely related to many cancers. In astrocytomas, although CDKN2A (p16INK4A protein) is often inactivated, there are still some controversial issues regarding the mechanism by which ...
Differential Expression of ADAM23, CDKN2A (P16), MMP14 and VIM Associated with Giant Cell Tumor of Bone
(Journal Of Cancer, 2015)
Though benign, giant cell tumor of bone (GCTB) can become aggressive and can exhibit a high mitotic rate, necrosis and rarely vascular invasion and metastasis. GCTB has unique histologic characteristics, a high rate of ...